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Activated members of MAPKs can trigger intracellular pathways in glial cells leading to the release of glial mediators including proinflammatory cytokines and ROS which contribute to pain potentiation through peripheral and central sensitization 26

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Interestingly, by similarly inhibiting specific pathway components, it was shown that this signaling was dependent on G subunits derived from D2Rs G i/o , stimulating PLC activity

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