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In HG-treated HK-2 and HFD/STZ-induced C57BL/6J mouse models, Finerenone (FIN) was found to restore mitophagy, reduce oxidative stress and apoptosis, and alleviate renal tubular injury through the PI3K/Akt/eNOS signaling pathway, thus exerting a certain therapeutic effect on DKD [139]

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3,4 Thus, by preventing the degradation of incretin hormones, DPP-4 inhibitors prolong the physiologic effects of these molecules

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