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Research conducted on rats showed that this synthetic peptide can counteract the effects of cuprizone, a drug that induces demyelination of nerve fibers and mimic the damage caused by multiple sclerosis, by reducing nerve damage [10]

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These findings provide compelling evidence for the role of epigenetic regulation of CCL11 and suggest that supplemental B12 has potential as an epidrug to treat neurodegenerative conditions associated with aberrant CCL11 expression

Our philosophy acknowledges that modern lifestyles, dietary patterns, and certain medications can all contribute to suboptimal B12 status, regardless of age or overall health