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On the contrary, addition of superoxide dismutase to EGCG treated esophageal squamous cancer cells increased the inhibitory effect by stabilizing EGCG [78]

It aids in removing harmful substances and ensuring the efficient functioning of vital organs

It also modulates AMPK/mTOR signaling, influencing cardiomyocyte metabolism and autophagy, and thus CH regulation (202, 228)

TIGAR is required for efficient intestinal regeneration and tumorigenesis

However, silencing HO-1 increases the growth inhibition of hepatocellular carcinoma (HCC) cells after ferroptosis triggering, suggesting that HO-1 also has an anti-ferroptosis effect (36)