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These data demonstrate that HIV-1 gp120 triggers a pro-inflammatory response and neuropathology via CXCR4-NLRP3-dependent pyroptosis (Fig

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First, they found that glucose production, G6pc expression, and Pck1 expression were significantly lower in NNMT -overexpressing hepatocytes with Sirt1 inhibition than in NNMT -overexpressing hepatocytes without Sirt1 inhibition and that Sirt1 overexpression rescued the suppression of G6pc expression and Pck1 expression induced by NNMT knockdown [14]

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