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Initial studies demonstrated that phoenixin stimulates gonadotropin-releasing hormonedependent luteinizing hormone secretion, implicating it in reproductive regulation ( Phoenixin signals primarily through the G proteincoupled receptor GPR173, although aspects of its signaling biology remain incompletely defined
Using a model of induced DSBs in an endogenously CCNE1 overexpressing cell line (Tang et al., 2013), we found increased DSBs upon IDH1i treatment, which was rescued by KG and L-carnitine ( Fig
Lv SY, Chen WD, Wang YD
A study conducted at Peking University entitled Glutathione Augmentation: An Indicator of Cellular Health followed participants who received glutathione through different administration routes
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