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It took decades of pharmaceutical research and billions of dollars in development

Indeed, GLUTag cells express the IL-6 receptor, and IL-6 treatment increases GLP-1 secretion in a dose-dependent manner, where acute IL-6 treatment increases GLP-1 exocytosis in a JAK2-STAT3-dependent manner, and chronic IL-6 treatment increases GLP-1 content and glucose uptake in a sodium glucose transporter 1-dependent manner in the L cell (Ellingsgaard et al., 2011)

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This change reduces the metabolic strain associated with insulin resistance and sets the stage for sustained metabolic health even if treatment eventually ends