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Recent research specifically targeting the pathophysiology of Long COVID and ME/CFS has cemented the role of oxidative stress in these conditions
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Biomechanical forces in atherosclerosis-resistant vascular regions regulate endothelial redox balance via phosphoinositol 3-kinase/akt-dependent activation of Nrf2
Both genotypes produce similar deficiencies of -globin, but they have different implications for the children of affected individuals, who are at risk of clinically significant -thalassemia (HbH disease or hydrops fetalis) only when at least one parent has the / haplotype
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