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While the molecular mechanisms of APAP hepatotoxicity are diverse and include different players, including innate immune response, in this review we will focus on the mitochondriallysosomal axis, which has emerged as a novel liason in several pathologies, including lysosomal storage diseases (Torres et al., 2017a), to pinpoint its role in APAP-mediated hepatotoxicity

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Restoring glutathione availability may lower oxidative burden, but clinical translation depends on concurrent micronutrient repletion (selenium, zinc, iron) and resolution of the primary inflammatory driver

GLP-1 is a naturally occurring hormone in the body that plays a crucial role in regulating blood sugar levels by promoting insulin secretion

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