glutathione cancer cell Application of depletion in therapy: Enhanced ROS-based therapy, ferroptosis, and chemotherapy Inhibition of Glutamate‐to‐Glutathione Flux Promotes

10.1016/0006-291X(83)91789-8

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Transport across the BBB may be facilitated by sodium-dependent transport system [76] or converting cysteine to cystine, which is subsequently exchanged with intracellular glutamate via the cystine-glutamate antiporter, and reduced back to cysteine for GSH production [115, 116]

The sulfhydryl (thiol)-containing tripeptide glutathione (GSH) is synthesized and maintained at high concentrations in virtually all cells and is one of the principal mechanisms by which reactive oxygen species (ROS) are eliminated [3, 7, 8]