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These include nutritional guidance, physical activity, emotion regulation skills, and self-monitoring, which are core components of both post-bariatric and general obesity programs
This pro-ferroptotic role of NO provides the rationale for using NO-releasing drugs to induce ferroptotic cell death in CRC (Tables 2, 3)
This month, two new studies help close the loop: Study #1 (Lancet eBioMedicine, July 2025): Longitudinal plasma proteomics in ~100 humans before and after metformin revealed 23 consistently altered proteins
Endocrinol Res Pract
Metformin further enhances oxidative stress resistance via upregulation of Klotho and GDF-15, promoting mitochondrial efficiency and anti-senescence signaling (Zhang et al., 2012)
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