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c-Jun N-terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)-dependent pathway leading to inactivation of intramitochondrial Src
However, some controversy prevails on the optimal dose
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This PlexusDx Education Hub explainer starts with that native signal, then builds up to the medications and the safety profile that share it
![Noncovalent Binding between Guanidinium and Anionic Groups: Focus on Biological- and Synthetic-Based Arginine/Guanidinium Interactions with Phosph[on]ate and Sulf[on]ate Residues](https://pubs.acs.org/cms/10.1021/cr040603j/asset/images/medium/cr040603jf1.gif)
