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[DOI] [PMC free article] [PubMed] [Google Scholar] 88.Zhu H.L., Zhang Y.S., Shi Z.S., Lu D., Li T., Ding Y., Ruan Y., Xu A
Our understanding of obesity since the discovery of leptin has revolved around the idea that BMI has a genetic basis, with approximately 4070% of the variation in BMI in the population being attributable to heritable factors [9], and that the majority of genetic variation associated with BMI is in the central nervous system (CNS) [10]
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Collectively, these two studies elucidate the role of EAT in AF from two distinct dimensions: EAT heterogeneity (pathological complexity) and overall mass (early quantitative changes)