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The abundance of GSH may be a compensatory mechanism against exposure to mild oxidative stress in the normal-appearing liver of the Aldh2*2 KI mouse

A theoretical multi-target strategy might combine: NAD+ for ongoing metabolic support: sirtuin activation, DNA repair, mitochondrial maintenance, nutrient sensing recalibration Epitalon for periodic telomere maintenance: cyclic telomerase activation to preserve replicative capacity and delay senescence onset This combination would address at least seven hallmarks between the two interventions

AndjelkovicMBuha DjordjevicAAntonijevicEAntonijevicBStanicMKotur-StevuljevicJet al