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It aids in the regeneration and protection of nerve cells, which is vital for preventing neuropathy

That does not close the door on metabolic therapies and brain health, but it does argue against treating GLP-1s as a dementia-prevention shortcut

DOI: 10.1159/000341454 [Google Scholar] Xu WW, Guan MP, Zheng ZJ, Gao F, Zeng YM, Qin Y, et al

For all of us

63 Prolonged skeletal muscle inactivity promotes mitochondrial dysfunction, resulting in increase in mitochondrial ROS production and leading to protracted oxidative stress and disturbed redox signaling, 64 which can exacerbate IR through multiple pathways such as JNK and NF-B pathways (Fig