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Acsl4 deletion attenuated renal impairment and oxidative stress in the oxalate-induced AKI mouse model To clarify the significance of ferroptosis in oxalate-induced AKI, we first generated a mouse model with renal tubule-specific deletion of ACSL4 ( Acsl4 fl/fl Ksp Cre-ERT2 ) (Fig
The transcript mentions: Cancer Parkinsons disease Diabetes Liver disease Heart disease Chronic recurring ear infections There was also a study mentioned involving children with chronic recurring ear infections
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