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The therapeutic effects arise through three primary mechanisms: central appetite suppression via hypothalamic GLP-1R activation (reducing food intake by 20-35%), delayed gastric emptying through vagal pathways (enhancing satiety per meal), and glucose-dependent insulin secretion from pancreatic beta cells (improving glycemic control)

Twenty-seven studies reported adverse effects, which are summarized in Supplementary Table 3

Most side effects go away on their own as your body gets used to the medicine

This cohort was also used in Experiments 3 and 4

They include nuts, seeds, nut butters, and a few high-fat fruits, such as avocados and olives