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Cpt1a Promoted ROS-Induced Oxidative Stress and Inflammation in Liver Injury via the Nrf2/HO-1 and NLRP3 Inflammasome Signaling Pathway

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(5) However, treatment with buprenorphine should only be initiated in patients whom there is a low potential of precipitating withdrawal

To further characterize these interactions, we constructed a molecular interaction network (Figure 7C) revealed three functionally interconnected modules: ( Consistent with multi-omics findings, KEGG pathway analysis identified glutathione metabolism as the most significantly perturbed shared pathway (Figure 8A), exhibiting coordinated changes between upregulated antioxidant enzymes such as Gpx3 and Mgst1 alongside depleted glutathione derivatives, supporting redox imbalance as a key pathogenic feature