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The comparison favors carnitine's metabolic approach for sustainable, long-term energy metabolism enhancement, offering superior tolerability and broader energy-supporting benefits

Basic research supports this mechanism, demonstrating that fructose or HFD exposure induces mitochondrial structural abnormalities, mtDNA depletion, and ATP deficiency, ultimately impairing -oxidation and hepatocyte function

Well-controlled investigations involving comparative arms, molecular profiling, functional endpoints, and temporal analysis may clarify whether this regenerative synergy holds up under experimental rigor

and (iii) the GSH:hGSH ratio must reach a certain value

Reich R, Blumenthal M, Liscovitch M