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It has been widely reported that an exercise bout consisting of eccentric muscle contractions can result in substantial damage to the contractile unit, which often results in inflammation, loss of Ca 2+ homeostasis and possibly induction apoptosis as a result

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Consistent with the effects seen in rat PSCs, high glucose and TGF1 enhanced the proliferation and decreased the apoptosis of hPSCs under low glucose conditions, whereas GSH and SB431542 prevented these effects in the presence of high glucose (Fig