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Neuronal sensitivity to kainic acid is dependent on the Nrf2-mediated actions of the antioxidant response element

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Our recent study, on acetaminophen-induced toxicity in macrophages has also suggested that differential inhibition of mitochondrial respiratory enzymes is associated with altered ROS/NO homeostasis in mitochondria [53].Our recent publications have also discussed extensively the mitochondrial respiratory dysfunctions under increased oxidative stress conditions [23], [25], [38], [42], [43]

And, while this particular side effect also appears rare, an association is being established in studies of patients taking GLP-1 receptor agonists