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Moreover, LC treatment (300 mg/kg body weight per day) for 7 and 14 d caused significant reduction of the tissue lipid peroxidation and marked improvement of the antioxidant status in atherosclerotic rats 41 , and the long-term oral administration of LC was associated with a decrease of plaque cell proliferation and the severity of aortic atherosclerotic lesions in a rabbit model 22

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A deficiency in any of these nutrients can mean that homocysteine is not converted properly, resulting in elevated levels

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