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moreover, crossing SOD1 G93A mice with mice overexpressing Nrf2 selectively in astrocytes significantly delayed disease onset and extended survival, thus reversing the toxicity of astrocytes expressing human SOD1 G93A mutation to co-cultured MNs [266, 267]

Increases of 510 mmHg systolic are common, with peak effect at 4 hours post-dose

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Schematic of key metabolites and enzymes (red) downstream of SAM