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It has been reported that elevated expressions of vascular endothelial growth factor (VEGF) and interleukin 8 (IL8) induce the production of vascular endothelial cells in ectopic endometrial lesions and that anti-VEGF/VEGF receptor treatments suppress the development of endometriosis in animal models [12, 13]

For CTA and kaolin-intake studies, male Wistar rats (Janvier Labs, WistarRjHan:WI) were used

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