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Inflammation originating from adipose tissue is driven by caloric excess and organelle functional decompensation, leading to adipocyte hypertrophy, hypoxia, and macrophage infiltration, while concurrently activating the secretion of proinflammatory mediators such as tumor necrosis factor- (TNF-) and interleukin-6 (IL-6) from adipocytes (111)

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Saturated fats may also activate TLR4s, leading to the stimulation of the inflammatory pathways and cytokine release (34)

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