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The question raised by researchers was whether some smaller molecule, not as susceptible to breakdown as a polypeptide by the bodys enzymes, could still trigger activity even if it only fits into a part of the receptor

High concentration of GABA and high glutamate decarboxylase activity in rat pancreatic islets and human insulinoma

The complex, multi-factorial pathogenesis of SAH, with intertwined pathways, strongly suggests that single-agent therapies may be insufficient to achieve sustained clinical benefit [14,31]

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