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Preliminary evidence of attenuation of the disruptive effects of the NMDA glutamate receptor antagonist, ketamine, on working memory by pretreatment with the group II metabotropic glutamate receptor agonist, LY354740, in healthy human subjects

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There is no universal retesting schedule for total glutathione, F2-isoprostanes, oxidized LDL, or MPO

TMAO may induce defects in synaptic plasticity through the PERK signaling pathway mediated by ERS