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Abstract Multiple sclerosis is a severe demyelinating disease mediated by cells of the innate and adaptive immune system, especially pathogenic T lymphocytes that produce the pro-inflammatory cytokine granulocyte-macrophage colony stimulating factor (GM-CSF)

Conversely, pathogenic Th17 cells express Tbx21, Ifng, Il23r, Gzmb and Csf2 ( As a consequence of being exposed to the aforementioned cytokines, these T lymphocytes can produce IL-17A, IL17F, IL-21, IL-22 ( One important subtype of lymphocytes are T helper (Th) cells that produce granulocyte-macrophage colony-stimulating factor (GM-CSF), initially hypothesized to be a growth factor for cells of the myeloid lineage

Chaibunruang, A

PMID: 16352683

1A) and the cellular level (Fig