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Nevertheless, other inhibitors, including GLS1/GLS2 inhibitor compound 968 and G6PD inhibitor 6-aminonicotinamide (6-AN) were failed to induce the above alterations in VRK2-KO and VRK2-KD cells (Fig

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To investigate the impact of shifting from cystine-driven to GSH-driven cysteine supply in cancer cells in an unbiased manner, we performed a multifunctional approach to pharmacological screening (MAPS) with a library of 240 metabolic inhibitors (Fig

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