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Although SRs have not been directly linked to the development of fever, they influence proinflammatory signaling pathways, such as nuclear factor kappa-light-chain-enhancer of activated B cells (NF-B) and JAK/STAT, that regulate the febrile response (383)

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HPR enzymes are important in photosynthesis because the knockout mutant of HPR1 shows compromised photosynthetic performance and growth in ambient air, with additive effects observed in the hpr1 hpr2 double and hpr1 hpr2 hpr3 triple mutants 16,17