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Early clinical trials of glutamine antagonist metabolites have revealed unacceptable systemic toxicity, highlighting the need for more selective approaches to disrupt glutamine metabolism in cancer patients [225]

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Smith PD, Smythies LE, Mosteller-Barnum M et al (2001) Intestinal macrophages lack CD14 and CD89 and consequently are down-regulated for LPS- and IgA-mediated activities

Glutamine also caused a significant increase in plasma insulin levels, particularly in obese subjects

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