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Here is the literal biology: GHK-Cu activates Wnt/beta-catenin signaling to restart dormant follicles, suppresses TGF-beta1 to prevent premature regression, stimulates VEGF for perifollicular blood supply, and protects dermal papilla cells from programmed death (Pickart & Margolina, 2018)

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It turns out that even in mice that lack these receptors, fat loss takes place when AOD9604 is administered Though the beta-3-adrenergic receptor likely plays a role in fat loss secondary to AOD9604, at least one other mechanism must be in play as well

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