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Furthermore, the increased expression of HO-1 in individuals suffering from heart failure serves to mitigate the adverse effects of pathological left ventricular remodeling, diminish myocardial hypertrophy, minimize oxidative stress, and abate inflammatory activation [96]

The proposed mechanism for CM ingestion is firstly dependent on the citrulline component via the L-arginine-NO pathway, such that following NO synthesis the smooth muscle may relax leading to vasodilation (Vanhoutte et al

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Addressing these mood imbalances is vital for overall healing