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Ethanol consumption induces the accumulation of CYP2E1 (130), which is a major contributor to ethanol-induced ROS production in the liver

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While cell lines have informed (sub)cellular pharmacology, they do not allow endogenous GPCRs to be interrogated, nor do they replicate the tissue environment where heterogeneity and cell-cell interactions are critical for shaping signaling responses 3,4,5,6

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