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The supernatant was reacted with 10 mM acid ninhydrin solution dissolved in 80% ethanol and 0.2 mM citrate buffer (pH 5.0) using the diluent solution (water: n-propanol = 1:1)

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Further, these data suggest that by maintaining circulating triglycerides, GSH synthesis in the liver potentially supports fat stores in the body

Inhibiting DHODH activity leads to a reduction in COQ10 levels, compromising antioxidant capacity and increasing susceptibility to lipid peroxidation and ferroptosis [13]

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