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In short, these findings underscored that increased autophagy and improved mitochondrial dysfunction was involved in the cardioprotective effects of novel antidiabetic drugs (Fig

Consistently, intestinal-specific overexpression of TXNIP abolished metformin-induced improvements in glucose tolerance, while pharmacological inhibition of GLUT1 similarly negated metformins glucose-lowering effects

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Nicotinate-curcumin ameliorates cognitive impairment in diabetic rats by rescuing autophagic flux in CA1 hippocampus