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Your healthcare team can also review whether your current GLP-1 dose remains appropriate or whether adjustment might improve tolerability whilst maintaining therapeutic benefit

Ultimately, these findings suggest that SAAR-mediated increase in glucose tolerance is the result of liver responses to maintain SAA homeostasis via serine synthesis

The consistency of results across both diabetes and obesity populations suggests that dual agonism provides robust metabolic benefits that extend beyond glucose control to address the full spectrum of metabolic dysfunction

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"One of the [adverse] effects that people will talk about