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Accessed August 14, 2025

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Chronic hyperglycemia-induced ROS overproduction, primarily derived from mitochondrial ETC leakage and NOX activation, leads to eNOS uncoupling due to BH4 oxidation, reducing NO bioavailability and impairing vasodilation (Giacco and Brownlee, 2010)

In addition, carthamin yellow therapy suppressed the accumulation of Fe 2+ and ROS and restored the levels of ACSL4, TFR1, GPX4, and FTH1 proteins in the brain (Guo et al., 2021)

Pregnancy: The weight of the fetus and hormonal changes can lead to swollen veins