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GLP-1 is a promising target for the treatment of depression because impaired neurogenesis ability and the reduction of neuronal differentiation leads to multiple depressive pathological symptoms

Dosing is carefully titrated under physician supervision, starting with low doses and adjusting based on individual response

Or placebo-treated patients in clinical trials may have different baseline risk profiles than real-world patients on alternative medications

Clinical evidence of antidepressant effects of insulin and Anti-Hyperglycemic agents and implications for the pathophysiology of Depression-A literature review

Patients with acute kidney injury (AKI) or unstable renal function at baseline were excluded